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BMC Cell Biology

Featured article: Cellular response to environmental stress

Sampuda et al. 2017

Accumulation of damaged or misfolded proteins in C.elegans in response to environmental stressors is the precursor to rapid formation of stress induced nuclear granules, which may be part of a novel protein quality control system.

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2. RESEARCH ARTICLE

   Cladosporol A triggers apoptosis sensitivity by ROS-mediated autophagic flux in human breast cancer cells

   Mytre Koul, Ashok Kumar, Ramesh Deshidi, Vishal Sharma, Rachna D. Singh, Jasvinder Singh, Parduman Raj Sharma, Bhahwal Ali Shah, Sundeep Jaglan and Shashank Singh

   Published on: 20 July 2017

   The Erratum to this article has been published in BMC Cell Biology2017 18:27
3. RESEARCH ARTICLE

   The M-phase specific hyperphosphorylation of Staufen2 involved the cyclin-dependent kinase CDK1

   Rémy Beaujois, Elizabeth Ottoni, Xin Zhang, Christina Gagnon, Sami HSine, Stéphanie Mollet, Wildriss Viranaicken and Luc DesGroseillers

   Published on: 14 July 2017
4. RESEARCH ARTICLE

   External magnetic field promotes homing of magnetized stem cells following subcutaneous injection

   Yu Meng, Changzhen Shi, Bo Hu, Jian Gong, Xing Zhong, Xueyin Lin, Xinju Zhang, Jun Liu, Cong Liu and Hao Xu

   Published on: 26 May 2017
5. RESEARCH ARTICLE

   Adenylyl cyclase mRNA localizes to the posterior of polarized DICTYOSTELIUM cells during chemotaxis

   Satarupa Das, Joshua M. Parker, Can Guven, Weiye Wang, Paul W. Kriebel, Wolfgang Losert, Daniel R. Larson and Carole A. Parent

   Published on: 25 May 2017
6. RESEARCH ARTICLE

   ULK1 phosphorylates Sec23A and mediates autophagy-induced inhibition of ER-to-Golgi traffic

   Wenjia Gan, Caiyun Zhang, Ka Yu Siu, Ayano Satoh, Julian A. Tanner and Sidney Yu

   Published on: 10 May 2017

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Connexin26 mutations and KID syndrome

This micrograph shows a cell with a D50Y mutation in connexin26. Cx26 protein (red) and golgin-97 (green) colocalize more frequently than in wildtype cells. D50Y and I30N mutations result in the formation of aberrant hemichannels that might elevate intracellular calcium levels, which may contribute to the hyperproliferative epidermal phenotypes of KID syndrome.

Aypek et al. BMC Cell Biol. 17:5