Prostate Cancer, Nutrition, and Dietary Supplements (PDQ®)–Health Professional Version
SECTIONS
- Introduction
- Calcium
- Green Tea
- Lycopene
- Modified Citrus Pectin
- Pomegranate
- Selenium
- Soy
- Vitamin D
- Vitamin E
- Multicomponent Therapies
- Other Prostate Health Supplements
- Changes to This Summary (05/10/2018)
- About This PDQ Summary
- View All Sections
Changes to This Summary (05/10/2018)
The PDQ cancer information summaries are reviewed regularly and updated as new information becomes available. This section describes the latest changes made to this summary as of the date above.
Revised text to state that green tea polyphenol composition and the ratio of monomeric versus oligomeric catechins can vary widely, depending on processing and source of the tea leaves. Considering that (−)-epigallocatechin-3-gallate and other monomeric catechins interfere with in vitro assays and exhibit a wide range of biological effects, this indicates that the chemical factors responsible for the actual in vivohealth benefits of green tea are mostly unknown (cited Bisson et al. as reference 7).
Revised text to state that two meta-analyses examined the consumption of green tea and prostate cancer risk, with one meta-analysis including black tea (cited Guo et al. as reference 28).
Added text to state that a 2015 systematic review and meta-analysis of studies investigating dietary lycopene intake/circulating lycopene levels and prostate cancer risk found that when lycopene intake was higher, the incidence of prostate cancer was reduced (cited Chen et al. as reference 24). Similarly, a higher level of circulating lycopene was associated with lower prostate cancer risk. Likewise, a 2017 systematic review and meta-analysis evaluated lycopene dietary intake and circulating lycopene with prostate cancer risk. An inverse association between high levels of both circulating and dietary lycopene with prostate cancer risk was noted (cited Rowles et al. as reference 25).
Added Paller as reference 19.
This summary is written and maintained by the PDQ Integrative, Alternative, and Complementary Therapies Editorial Board, which is editorially independent of NCI. The summary reflects an independent review of the literature and does not represent a policy statement of NCI or NIH. More information about summary policies and the role of the PDQ Editorial Boards in maintaining the PDQ summaries can be found on the About This PDQ Summary and PDQ® - NCI's Comprehensive Cancer Database pages.
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