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Featured collection: Proceedings of the International Gap Junction Conference 2015

This article collections originated from the International Gap Junction Conference, held in Valparaiso, Chile, in March 2015. The collection contains 11 reviews of the structure, cell biology, pharmacology and other aspects of gap junction channels and hemichannels.

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Featured article: Recovering from cold shock

 This study follows the phases of death associated with cold shock in C. elegans. Cold shock of under 12 hours does not initially kill the worms but starts a progression over 4 days, after which they either successfully recover, or fail to do so and die. Worms lacking the G-protein coupled receptor FSHR-1 are resistant to acute death from longer cold shocks and are more successful in their recovery from shorter sub-lethal cold shocks.

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1. METHODOLOGY ARTICLE

   Hierarchical imaging: a new concept for targeted imaging of large volumes from cells to tissues

   Irene Wacker, Waldemar Spomer, Andreas Hofmann, Marlene Thaler, Stefan Hillmer, Ulrich Gengenbach and Rasmus R. Schröder

   Published on: 12 December 2016
2. RESEARCH ARTICLE

   Nuclear envelope structural defect underlies the main cause of aneuploidy in ovarian carcinogenesis

   Callinice D. Capo-chichi, Toni M. Yeasky, Elizabeth R. Smith and Xiang-Xi Xu

   Published on: 22 November 2016
3. RESEARCH ARTICLE

   A functional endosomal pathway is necessary for lysosome biogenesis in Drosophila

   Anne-Claire Jacomin, Marie-Odile Fauvarque and Emmanuel Taillebourg

   Published on: 16 November 2016
4. RESEARCH ARTICLE

   Expressed alleles of imprinted IGF2, DLK1 and MEG3 colocalize in 3D-preserved nuclei of porcine fetal cells

   Yvette Lahbib-Mansais, Harmonie Barasc, Maria Marti-Marimon, Florence Mompart, Eddie Iannuccelli, David Robelin, Juliette Riquet and Martine Yerle-Bouissou

   Published on: 1 October 2016
5. RESEARCH ARTICLE

   Loss of hif-1 promotes resistance to the exogenous mitochondrial stressor ethidium bromide in Caenorhabditis elegans

   Muntasir Kamal, Dayana R. D’Amora and Terrance J. Kubiseski

   Published on: 13 September 2016

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Connexin26 mutations and KID syndrome

This micrograph shows a cell with a D50Y mutation in connexin26. Cx26 protein (red) and golgin-97 (green) colocalize more frequently than in wildtype cells. D50Y and I30N mutations result in the formation of aberrant hemichannels that might elevate intracellular calcium levels, which may contribute to the hyperproliferative epidermal phenotypes of KID syndrome.

Aypek et al. BMC Cell Biol. 17:5